![]() ![]() Excessive renin leads to aldosteronism and potassium loss. Kidney Structure The kidneys are encased in complex layers of fascia and fat. ![]() Under certain circumstances sodium loss can potentiate both the overproduction and the unresponsiveness. The adrenal glands sit immediately superior to the kidneys within a separate envelope of the renal fascia. ![]() In the syndrome of juxtaglomerular hyperplasia with normal blood pressure, overproduction of renin may result from unresponsiveness of blood vessels leading to a lack of feedback inhibition by pressure rise. The adrenal gland (surrenal) is a hormone-producing organ located on both kidneys, which has little relationship with the kidney, contrary to popular belief. Progesterone may cause sodium loss and overproduction of renin and aldosterone while blocking their effects. In the non-salt-losing form of the adrenogenital syndrome of congenital adrenal hyperplasia without treatment, failure of feedback inhibition by Cortisol may result in overproduction of adrenocorticotropic hormone (ACTH) which, in turn, may lead to overproduction of progesterone. The adrenal glands are located just above the kidneys. ![]() In renovascular hypertension, angiotensin and aldosterone production may persist despite systemic hypertension. Adrenal insufficiency is a disorder that occurs when the adrenal glands don’t make enough of certain hormones. In the form of “primary” aldosteronism resulting from hyperplasia of all adrenal cortical tissue, overproduction of aldosterone persists in the absence of all known stimulatory factors. ![]()
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